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Ouabain and digoxin may induce increases in genetic expression of the proapoptotic BCL-2 family such as NOXA through activation of the JNK, GSK-3, and P38 pathways, partially mediating their senolytic effects, although this complete mechanism remains elusive [94]

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Furthermore, the B reg subsets expanded by Tα1 treatment display a suppressive activity reducing both IFN-γ and IL-17 production found in TLR7-treated MS patients-derived PBMC cultures (Giacomini et al., 2017)