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Taken together, disturbed Li homeostasis may contribute to early AD biology (potentially via GSK3β/β-catenin signaling and microglial/myelin vulnerability), but lithium is not an established AD therapy

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Another notable advancement is the emergence of “organs-on-chips” (OoCs) platforms, which faithfully replicate the dynamic microenvironment of human organs, facilitating in vitro assessment of systemic toxicity (Figure 3)